Informational Video I found–wanted to share

I think this is a good pic for GBSers to see as it shows the axon also and cell body and dendrites, I beleve the dendrites are the ones that will grow out new ones to make up for the ones that were killed off in the original GBS attack, This only happens when there is axon damage during the GBS,with the reserch I’ve read all GBS variants have Axon damage but the ones like AMAN which I believe involves a lot of the Axons. God Bless Dan

[QUOTE=wheelchairdan]I think this is a good pic for GBSers to see as it shows the axon also and cell body and dendrites, I beleve the dendrites are the ones that will grow out new ones to make up for the ones that were killed off in the original GBS attack, This only happens when there is axon damage during the GBS,with the reserch I’ve read all GBS variants have Axon damage but the ones like AMAN which I believe involves a lot of the Axons. God Bless Dan[/QUOTE]
Sorry–Here is the Site–DUH
[url]http://www.webmd.com/hw-popup/Neuron[/url]

I know most of these mayhave been seen by you all but here they are if interested. God Bless –Dan

[url]http://www.angelfire.com/home/gbs/[/url]

[url]http://www.jsmarcussen.com/gbs/print/residual2.htm[/url]

[url]http://www.geocities.com/CapitolHill/4277/me-restore.html[/url]

[url]http://www.nature.com/nm/journal/v6/n7/full/nm0700_738.html[/url]

[QUOTE=ali]Dan,

I have never seen the nature.com article before, I started reading it, but my brain wasnt taking much more in towards the end, so I will carry on a little later. Thank you for that, It is very interesting.

(Do you get ‘The Communicator’ newsletters? Have you read the “Whats in a Name?” article from last year?)[/QUOTE]

Hi Ali. I never saw this article,do you have it? maybe you could scan and send so we can read, Thanks–P.S. I did look on the net and did come up with this from the Communicator 2004. I’ll tell you the end part about
Axonal regeneration sounds just like Post Polio Doesn’t it–BECAUSE IT IS THE SAME!!
God bless— Dan

Sunday, March 14, 2004
GBS Newsletter – The Communicator Spring 2003
Residual Effects Following Guillain-Barre
Gareth J. Parry, MD Professor of Neurology, University Of Minnesota

Guillain-Barr~ syndrome is a disorder whose excellent prognosis is invariably emphasized. Widely accepted figures suggest that 75%-85% of patient make a complete recovery. However, many of my patients have complained to me of minor but annoying persistent symptoms continuing for years after the initial paralytic event. Although I have made no systematic study of the proportion of patients with these residual complaints it is certainly more than the 15%-25% that the figures in the literature would suggest. The great majority of studies of the ultimate outcome in GBS are based on telephone interviews or retrospective chart reviews and seemingly minor complaints may have been missed or disregarded. Thus, patients are often asked if they have returned to their previous work or other previous activities but they may not have been asked whether they have more difficulty performing their former activities. A note of caution was sounded in one small study from Dr. J. McLeod and his colleagues in Australia (J. Neurol Sci 1976; 27:438-443) who objectively evaluated a small group of 18 recovered GBS patients and found that half of them had objective residual neurological abnormalities. Even then the residual abnormalities were considered to be significant in only four patients. A recent important paper from Dr. I.S.J. Merkies and colleagues in Holland (Neurology 1999; 53:16481654) has established that residual effects from both GBS and CIDP are much more common than has been generally reported and that seemingly minor neurologic abnormalities may still result in annoying disabilities. The study used a validated index of fatigue severity to assess residual disability. It included 83 patients who had suffered from GBS an average of five years previously. About 80% of these patients experienced fatigue that was considered severe enough to interfere with their life despite the fact that the majority had normal strength or only minor weakness. They noted also that the fatigue did not seem to improve over time; the fatigue index score was the same in patients in whom many years had elapsed as it was in patients whose acute illness had occurred only 6-12 months previously. This paper provides sound scientific support for the validity of the observations of my patients who regularly complain of fatigue even when they have returned to all or most of their former activities and who are working full time at their former jobs. Although their strength may be normal when they are examined in the doctor’s office they are clearly unable to sustain the same level of physical activity that they had performed prior to their GBS.
A second under-appreciated symptom that may persist for many years is pain. Certainly, severe disabling pain is very rare. However, a number of my patients complain of persistent discomfort in their feet. The discomfort may take the form of annoying paresthesias (tingling) or there may be a vague aching discomfort. Occasionally there is more severe burning or stabbing pain. The symptoms have the same characteristics as typical neuropathic pain in that they tend to be worse in the evening or at night and are particularly annoying following days during which the patients have been up on their feet a lot. The discomfort is not particularly responsive to analgesics but usually does respond to antiepileptic drugs such as gabapentin, or antidepressant drugs such as amitriptyline, drugs typically used in the treatment of neuropathic pain. However, these medications have to be taken on a daily basis to be effective and one problem with deciding whether to treat this residual symptom is that the discomfort is usually rather mild. Thus, patients may be daily irritated by their symptom but be reluctant to take a drug every day for a symptom that significantly bothers them only once or twice a month. I have seen no mention in the medical literature of this phenomenon. It is possible that I see a highly selected group of patients in my practice who had initially been more severely affected and that the prevalence of this annoying residual symptom is much higher in my patients than in the usual population of recovered GBS patients. I would be most interested to learn whether the group of patients reported by Merkies and colleagues also suffered from minor persistent discomfort.
The basis for both of these seemingly minor residual symptoms (fatigue and pain) is probably axonal degeneration. During the acute illness the predominant underlying pathology in most patients is segmental demyelination, a completely reversible phenomenon. However, some degree of axonal degeneration is almost invariable and in some patients it is severe. As recovery occurs function is restored by a number of mechanisms. Axonal regeneration of motor axons probably plays very little role in restoration of function except in the more severe cases. Rather, surviving axons send out small branches called collateral sprouts that restore the nerve supply to those muscle fibers whose nerves have been damaged. This process of collateral sprouting is very effective at restoring strength to a muscle but the efficiency of the muscle suffers the muscle must work harder to achieve its goals. Thus, fatigue may result even when there appears to be full restoration of strength. On the sensory side, even a small number of damaged sensory axons may be sufficient to generate spontaneous discharges that are registered as pain or discomfort.
It is entirely appropriate that the good outcome of GBS should be emphasized during the acute illness. During this time, the patient is losing control of many motor functions, sometimes including life-preserving functions, and constant reassurance from the attending physicians plays a vital role in the recovery process. However, it is equally important to be aware that residual problems are experienced by “recovered” GBS patients. Acknowledgement that such residual problems exist will go a long way towards helping patients deal with the frustration of their incomplete recovery.
More research is needed to discover an effective treatment for the residual fatigue. In addition, since these persistent symptoms are probably related to the degree of axonal damage that occurs at the time of the initial attack, we also need to continue to strive for earlier and more effective treatment of the acute stage of the disease so that these residual problems are minimized.

[url]http://www.ncbi.nlm.nih.gov/sites/entrez?Db=pubmed&Cmd=ShowDetailView&TermToSearch=17351693&ordinalpos=7&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_RVDocSum[/url]

[url]http://books.google.com/books?id=L2TJDwy0l_0C&pg=PA41&lpg=PA41&dq=renewed+weakness+axonal+guillain+barre+syndrome&source=web&ots=3dIESe6lXE&sig=vMgFvRJVLnm2ho0J0w7Trez1gk0#PPP1,M1[/url]

[url]http://www.ncbi.nlm.nih.gov/sites/entrez?Db=pubmed&Cmd=ShowDetailView&TermToSearch=11328201&ordinalpos=33&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_RVDocSum[/url]

[url]http://www.ncbi.nlm.nih.gov/sites/entrez?Db=pubmed&Cmd=ShowDetailView&TermToSearch=17290096&ordinalpos=11&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_RVDocSum[/url]

[QUOTE=peggy80]Thanks for Google video, Very informattive and well presented. Do you know where I can esearch to get more information on GBS and MG (myasthenia gravis}? I have both autoimmunes and another, pernicious anemia.

Peggy[/QUOTE]
Hi Peggy, I think the best way to research is to GOOGLE The form of GBS you have and maybe together with the MG, Good luck researching and God Bless

Understanding Guillain-Barré syndrome and central nervous system involvement

Schoool of Psychology, Massey University, Palmerston North, New Zealand. [email]MaryGregory566@hotmail.com[/email]

Guillain-Barré syndrome is a rare neurological disease that causes paralysis and may necessitate hospitalization for some patients in its acute stages. It primarily affects the peripheral nervous system, though recent research has shown that for some patients, the central nervous system is involved. The acute phase often requires intensive care services. Recognition is growing that recovery is not as smooth and free of symptoms as previously thought. Following “recovery” some people endure long-term residual symptoms, such as fatigue and pain. Nursing input can be of value by providing support, information, explanations, and empathy to reassure patients and families. A greater understanding of the nature and course of the disease and its ramifications can lead to more effective nursing management and a faster rehabilitation process.

PMID: 16175926 [PubMed – indexed for MEDLINE]

How Guillain-Barre patients experience their functioning after 1 year.

Department of Neurology, Jeroen Bosch Hospital, ‘s-Hertogenbosch, the Netherlands. [email]r.bernsen@jbz.nl[/email]

OBJECTIVE: To analyze how the patient himself perceives his physical and social situation 1 year after Guillain-Barre syndrome (GBS). MATERIAL AND METHOD: The Dutch patients who participated in an international multicenter trial were asked to complete a self-administered questionnaire containing questions on their physical status at homecoming and at 12 months, as well as questions dealing with various aspects of their social condition. RESULTS: Ninety patients participated. Up to 72% had sensory disturbances and loss of power in part of the arms and up to 89% in part of the legs at homecoming. At 12 months, a significant improvement had occurred, but residua were perceived in 36 and 67%, respectively. The residua ranged from irritating to seriously disturbing in up to 49%, and only 33% felt completely cured. Furthermore, 32% had changed their work due to GBS, 30% did not function at home as well as before and 52% had altered their leisure activities. CONCLUSION: One year after the onset of GBS, a considerable number of patients still perceived a decrease of power and sensation with an often disturbing effect. GBS had an evident impact on daily life and social well-being. Copyright Blackwell Munksgaard 2005.

PMID: 15932357 [PubMed – indexed for MEDLINE]

Impairment in Guillain-Barré syndrome during the first 2 years after onset: a prospective study.Forsberg A, Press R, Einarsson U, de Pedro-Cuesta J, Widén Holmqvist L; Swedish Epidemiological Study Group.
Centre for Rehabilitation Research, Orebro, Sweden. [email]anette.forsberg@orebroll.se[/email]

OBJECTIVES: To provide a comprehensive description of impairment in patients with Guillain-Barre syndrome (GBS) in Sweden during the first 2 years after disease onset. METHODS: In this prospective multi-centre study, 42 patients, mean age 52 years, were evaluated at 2 weeks, 2 months, 6 months, 1 year and 2 years. Evaluations made use of validated, reliable measures of muscle strength, grip strength, finger dexterity, balance, facial-muscle function, respiratory function, gait, motor performance and sensory examination, and included patients’ own assessments of pain, fatigue and paraesthesia. RESULTS: Mechanical ventilation was required in 21% of patients. At 2 weeks, 1 year and 2 years after GBS onset: 100%, 62% and 55% of patients had submaximal overall muscle strength; 98%, 38% and 31% subnormal grip strength; and 38%, 14% and 12% affected facial-muscle function. At the same time points, 62%, 10% and 7% of patients were unable to walk 10 m independently; and affected sensation was detected in 93%, 55% and 52%. CONCLUSIONS: Recovery occurred mainly during the first year after onset. At 2 years, motor impairment and sensory impairment were each still detectable in more than 50% of patients. We conclude that residual impairment is significant, somatically widespread and, likely, persistent.

PMID: 15546603 [PubMed – indexed for MEDLINE]

What Is the Long-Term Outlook for Those With Guillain-Barre Syndrome?

Guillain-Barre syndrome can be a devastating disorder because of its sudden and unexpected onset. In addition, recovery is not necessarily quick. As noted above, patients usually reach the point of greatest weakness or paralysis days or weeks after the first symptoms occur. Symptoms then stabilize at this level for a period of days, weeks, or, sometimes, months. The recovery period may be as little as a few weeks or as long as a few years. About 30 percent of those with Guillain-Barre still have a residual weakness after three years. About 3 percent may suffer a relapse of muscle weakness and tingling sensations many years after the initial attack.

Where Can I Go for More Information?

The National Institute of Neurological Disorders and Stroke conducts and supports a wide range of research on neurological disorders, including Guillain-Barre syndrome.

For information on neurological disorders or research programs funded by the National Institute of Neurological Disorders and Stroke, contact the Institute’s Brain Resources and Information Network (BRAIN) at:

BRAIN
P.O. Box 13050
Silver Spring, MD 20911
(800) 352-9424

Ali, Maybe you should call this institute and ask what takes place with this 3% of post GBS people and if this is the same as what happens with Post Polio people and maybe get something in writing for your presentation this fall. God Bless

Late effects of other disabilities

An article, Post-Everything Syndrome, appeared in the American magazine, New Mobility (September 2000). Author, Bonnie Moulton, discusses how polio survivors have raised medical and community consciousness about the late effects of all disabilities. She writes, We should give credit where credit is due. Polio survivors – inarguably the largest, most vocal and most politically active disability group in history – have fought to get health care providers, insurance companies and policy makers to recognise that for millions, the experience of polio didn’t end with recovery from the initial illness. The model of self advocacy they created has extended far beyond any single disability. She goes on to describe how survivors of spinal-cord injury, cerebral palsy, spina-bifida, Guillain-Barre syndrome, long term amputation and other physical disabilities considered to be static are experiencing late effects that seem like early ageing. As you will recall from the Network’s booklet Helping polio Survivors Live Successfully with the Late Effects of Polio, some polios develop post-polio syndrome which would seem to be unique to polio survivors. Most polios develop late effects due to the wear and tear of living with a compromised body. It is this that causes problems for people with other physical disabilities. Whatever the causes of their symptoms the different groups’ experiences are often remarkably similar. Pain tops the list, followed by increased fatigue, decreased endurance, increased spasticity and new muscle weakness … protocols that have helped polio survivors, including a transition to a ‘conserve to preserve’ lifestyle may be equally valuable to someone with spinal cord injury or spina bifida. It means that people with Guillain-Barre syndrome have every reason to print out an article from a post-polio Web site and tell their family care physician, ‘This sounds like me! Let’s try some of these things’. The stories in the article will sound familiar to polio survivors. Ken, aged 51, with cerebral palsy, says, You slowly, or sometimes quite suddenly, realize that you can’t do things you used to do easily. Then you realize that you’re not really THAT old. That’s the frightening part. Steve, aged 59, who has spinal injury was told by his physiotherapist ‘You have two speeds now, slow and stop’ … I can’t do six things in one day anymore … As I’ve grown older I’ve learnt to make sure that I don’t make too many demands on myself. As I get better at doing that, I’ve begun to be at peace. The author argues that people with long-term disabilities are more alike than different so they should be a community rather than a collection of enclaves. To have access to services and supports we’ll need as we age, we need to build coalitions. Moulton has much to say about the failure of medicine in handling chronic conditions. She quotes one doctor who recommends that people with late effects of disability consult a specialist in rehabilitation medicine or in sports medicine, Why sports medicine? Once you’ve had a disability for 30 or 40 years, the analogy that you’re doing a marathon (every day!) doesn’t seem that far off. For this we pay a price. ‘There is no doubt that the presence of a disability causes a person to expend more physical, emotional and mental energy every day than a person who does not have a disability’, confirms Renee Kirkby, an amputee for 30 years

Health officials convinced the Chinese to rename the bulk of their polio to Guillaine Barre Syndrome (GBS). A study found that the new disorder (Chinese Paralytic syndrome) and the GBS was really polio . After mass vaccination in 1971, reports of polio went down but GBS increased about 10 fold…….In the WHO polio vaccine eradication in the Americas, there were 930 cases of paralytic disease—all called polio. Five years later, at the end of the campaign, roughly 2000 cases of paralytic disease occurred—but only 6 of them were called polio (41). The rate of paralytic disease doubled, but the disease definition changed so drastically that hardly any of it was called polio any more.”—Greg Beattie

[url]http://www.samcstl.org/mayo/parsexmldisplay.asp?xd=MM00374[/url]

I hope these are helping some. God Bless

[url]http://www.youtube.com/watch?v=h641luMEu8Y[/url]

[url]http://www.youtube.com/watch?v=-CB8aIxQVu0&NR=1[/url]

Hello all, This Video is a personal story that we should all see –it shows how family and God play a role in recovery.
I think it is very important that family be there everyday–even every minute with someone with GBS especially if they are Totally Paralyzed and on a vent for this is a time they need support the most and this is a time you can watch the treatment that they get and make sure they are not being abused by the hospital staff–sadly this does happen more than you may think and in my case 30 years ago in a VA hospital while on active duty I with the help of the Swine flu Shot they gave me was in this Paralyzed State and was abuse BADLY evrything from Verbal, Physical, and yes Sexual, RAPED while paralyzed so don’t think it can’t happpen, keep a close eye on your family member or friend with this disorder or others disorders that can leave them Paralyzed.

[url]http://video.google.com/videoplay?docid=-5742062471201007689[/url]

Thanks Ali & Dawn,
It took years of treatment for me to talk about the abuse, I live with the thoughts more now that I am Totally Disabled from this GBS renewed weakness–all those thought came rushing back–BIG TIME.
I know you say thanks for your service to me but I don’t rank that–not by a long shot, See back then when I enlisted it was during peace time–No wars going on– also I received a OTHER THAN HONORABLE DISCHARE–because I went AWOL from the medical unit I was in after I got out from the VA hospital, I fought my own WAR and that was the GBS and the Neglect and Abuse but being a teen in the Army and having a messed up head from all that crap I took off and went home because I hated them ALL, I was gone for about 80 days and tried to committ suicide by smashing up my car– As you can see I didn’t succeed but anyway I tuned myself in at that time and was put in jail for No insurance while waiting for the MP’s, After returning they offered me a OTH discharge and I signed myself out with that–yes It cost losing all my benefits, I went on with life and like most after GBS went back to work and I left it all behind me–but after being hit with this Renewed GBS crap I had to give up my job I held for 24 years straight, Had to Cash in all my savings-401k-Kids Collage–and almost lost my house, Now I live off my SSDI–Thank God I was able to work long enough to get enough to skim by on now with SSDI, I did get ahold of the VA and they said they would give me Medical Help ONLY so they gave me a Electric hospital bed and a wheelchair but no money. I use my wifes Insurance because I can’t stand going into the VA hospital–I go into a panic attack. Well I spilled enough of my guts for everyone to see–I better stop–Thanks again–Dan

[url]http://video.google.com/videoplay?docid=-6473048817782768299[/url]

I think I will make mine and I’ll post it on here also.

Recurrent Guillain-Barré syndrome following influenza vaccine.Seyal M, Ziegler DK, Couch JR.
Two patients recovered from an attack of Guillain-Barré syndrome and then had a second attack of this disease, with a shorter latent period, following monovalent influenza vaccination. These cases suggest that an attack of Guillain-Barré syndrome may result in greater risk of future episodes of the syndrome in conjunction with exposure to influenza or other vaccinations.

PMID: 566873 [PubMed – indexed for MEDLINE]

[url]http://www.renewamerica.us/columns/janak/070722[/url]

[url]http://content.lib.utah.edu/cdm4/item_viewer.php?CISOROOT=/ehsl-shw&CISOPTR=37&CISOBOX=1&REC=8[/url]

Notice in this video how being tired can worsen symptoms,this is very true with us with Post GBS symptoms also. God Bless you all

[url]http://content.lib.utah.edu/cdm4/item_viewer.php?CISOROOT=/ehsl-shw&CISOPTR=29&CISOBOX=1&REC=15[/url]

I guess we need to watch out how well we cook our chicken. Good video to share with new people of what GBS does. God Bless

[url]http://www.youtube.com/watch?v=LHmE26S6tkk&mode=related&search=[/url]

Diet could help,My doctor has me on a diet much like this one–BUT like most I break the rules, Check it out and God bless.

[url]http://www.youtube.com/watch?v=BVn-jmCi4zI&mode=related&search=[/url]

[url]http://www.youtube.com/watch?v=InwsLaTvhzY&mode=related&search=[/url]

[url]http://www.youtube.com/watch?v=vnGNZ6RDjlg&mode=related&search=[/url]

The insufficient reinnervation sounds just like studies of POST POLIO–GOOGLE IT AND SEE!!

Muscle Nerve. 2005 Jan;31(1):70-7. Links
Electrophysiological signs of permanent axonal loss in a follow-up study of patients with Guillain-Barré syndrome.Dornonville de la Cour C, Andersen H, Stålberg E, Fuglsang-Frederiksen A, Jakobsen J.
Department of Neurology, Aarhus University Hospital, Nørrebrogade 44, 8000 Aarhus C, Denmark. [email]lacour@akhphd.au.dk[/email]

The neurophysiological mechanisms for persisting impairment of motor function after Guillain-Barre syndrome (GBS) were assessed in 37 unselected patients 1-13 years after diagnosis. For evaluation of reinnervation and axonal loss, macroelectromyography (macro-EMG) including measurement of fiber density (FD) was performed. Data from neuropathy symptom score, neuropathy disability score, nerve conduction studies, and quantitative sensory examination were ranked and summed to a neuropathy rank sum score (NRSS). The isokinetic muscle strength at the ankle was measured. Signs of axonal loss with increase of either macro motor unit potential (macro-MUP) amplitude or FD occurred in 76% of patients. The macro-MUP amplitude correlated with muscle strength and with NRSS. Patients with evidence of residual neuropathy had increased macro-MUP amplitude and FD as well as decreased muscle strength compared to patients without evidence of residual neuropathy. We conclude that axonal loss takes place in a substantial number of GBS patients and is associated with permanent muscle weakness caused by insufficient reinnervation. Possible patterns of pathology are discussed in relation to the macro-EMG findings.

PMID: 15543551 [PubMed – indexed for MEDLINE]
God bless Dan

Check this study out–My doctor told me the same thing and said watch out for meds that affect the central nervous system–They gave me Meds after my gall bladder surgery that affected my CNS and thet almost lost me in recovery, I spent 6 days in the hospital.

Rehabil Nurs. 2005 Sep-Oct;30(5):207-12.Links
Understanding Guillain-Barré syndrome and central nervous system involvement.Gregory MA, Gregory RJ, Podd JV.
Schoool of Psychology, Massey University, Palmerston North, New Zealand. [email]MaryGregory566@hotmail.com[/email]

Guillain-Barré syndrome is a rare neurological disease that causes paralysis and may necessitate hospitalization for some patients in its acute stages. It primarily affects the peripheral nervous system, though recent research has shown that for some patients, the central nervous system is involved. The acute phase often requires intensive care services. Recognition is growing that recovery is not as smooth and free of symptoms as previously thought. Following “recovery” some people endure long-term residual symptoms, such as fatigue and pain. Nursing input can be of value by providing support, information, explanations, and empathy to reassure patients and families. A greater understanding of the nature and course of the disease and its ramifications can lead to more effective nursing management and a faster rehabilitation process.

PMID: 16175926 [PubMed – indexed for MEDLINE]

The cause of PPS is unknown, but it is thought to be due to a distal ….. with constant denervation and reinnervation after paralytic polio. …
[url]www.ott.zynet.co.uk/polio/lincolnshire/library/trojan/anticholinesterases.html[/url] – 53k

Material and methods: Eighty-one former polio sufferers were, … process of denervation and reinnervation has been assumed as the most probable cause (3). …
pt.wkhealth.com/pt/re/obes/fulltext.00000132-199908000-00002.htm

[QUOTE=wheelchairdan]The insufficient reinnervation sounds just like studies of POST POLIO–GOOGLE IT AND SEE!!

Muscle Nerve. 2005 Jan;31(1):70-7. Links
Electrophysiological signs of permanent axonal loss in a follow-up study of patients with Guillain-Barré syndrome.Dornonville de la Cour C, Andersen H, Stålberg E, Fuglsang-Frederiksen A, Jakobsen J.
Department of Neurology, Aarhus University Hospital, Nørrebrogade 44, 8000 Aarhus C, Denmark. [email]lacour@akhphd.au.dk[/email]

The neurophysiological mechanisms for persisting impairment of motor function after Guillain-Barre syndrome (GBS) were assessed in 37 unselected patients 1-13 years after diagnosis. For evaluation of reinnervation and axonal loss, macroelectromyography (macro-EMG) including measurement of fiber density (FD) was performed. Data from neuropathy symptom score, neuropathy disability score, nerve conduction studies, and quantitative sensory examination were ranked and summed to a neuropathy rank sum score (NRSS). The isokinetic muscle strength at the ankle was measured. Signs of axonal loss with increase of either macro motor unit potential (macro-MUP) amplitude or FD occurred in 76% of patients. The macro-MUP amplitude correlated with muscle strength and with NRSS. Patients with evidence of residual neuropathy had increased macro-MUP amplitude and FD as well as decreased muscle strength compared to patients without evidence of residual neuropathy. We conclude that axonal loss takes place in a substantial number of GBS patients and is associated with permanent muscle weakness caused by insufficient reinnervation. Possible patterns of pathology are discussed in relation to the macro-EMG findings.

PMID: 15543551 [PubMed – indexed for MEDLINE]
God bless Dan[/QUOTE]

Please share any videos I posted with others–God bless

[url]http://www.youtube.com/watch?v=xmjX806GvV8[/url]

Glad Merk decided to stop push–but some goverment officials on merk payroll are pushing this vaccine–Must listen to this video. Do you have to get your children vaccinated–NOT!!!!!!!!!!!!!!!!!! Spread the word,don’t let our schools control your choices–God Bless

[url]http://www.youtube.com/watch?v=heDVDSRhOMs[/url]

[url]http://www.youtube.com/watch?v=heDVDSRhOMs[/url]

People who have had GBS are not to get this vaccine–and it DOES CAUSE GBS–This burns my ass that the military is doing the same crap to our soldiers with anthrax that they did to me SWINE FLU SHOT 30 years ago–line up and shut your mouth–we heard of soldiers getting sick, SHUT UP AND GET IN LINE or get court marshalled, So hear I sit in a wheelchair 30 years later and the VA craps on me and others like me. God Bless

[url]http://www.youtube.com/watch?v=KhZs1pfPkJI&NR=1[/url]

[url]http://www.youtube.com/watch?v=KhZs1pfPkJI&NR=1[/url]

[QUOTE=wheelchairdan]People who have had GBS are not to get this vaccine–and it DOES CAUSE GBS–This burns my ass that the military is doing the same crap to our soldiers with anthrax that they did to me SWINE FLU SHOT 30 years ago–line up and shut your mouth–we heard of soldiers getting sick, SHUT UP AND GET IN LINE or get court marshalled, So hear I sit in a wheelchair 30 years later and the VA craps on me and others like me. God Bless

[url]http://www.youtube.com/watch?v=KhZs1pfPkJI&NR=1[/url]

[url]http://www.youtube.com/watch?v=KhZs1pfPkJI&NR=1[/url][/QUOTE]

[url]http://www.youtube.com/watch?v=9PQqllRISpI[/url]

[QUOTE=wheelchairdan][url]http://www.youtube.com/watch?v=9PQqllRISpI[/url][/QUOTE]

[url]http://www.youtube.com/watch?v=t8W9cvCR-lE[/url]

[url]http://www.youtube.com/watch?v=c4lbq2bFC1Y&feature=related[/url]