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Anonymous
March 21, 2009 at 9:33 am

I agree with the others, you do need to tell your doctor that you felt better and did better with the previous amount of IVIG’S you were on. Our doctors need that feedback from us. He/she can’t look at us and know how we feel because in a sense syndrome means “I don’t know”.

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Anonymous
January 23, 2009 at 8:08 pm

Hello,
I have CIDP, the chronic longer lasting form of GBS and my feet/ankles would turn out and all of a sudden I’d realize that I’d be standing/walking on the side of my feet. This usually happened when I’d try to stand up from a sitting position. For me, it lasted for about a year and hasn’t happened again since. We’re like snowflakes though (gbs/cidp), all have the same thing but each of us is different. I never had to wear braces/AFO’S, sturdy ankle high boots worked for me.

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Anonymous
July 28, 2007 at 4:39 pm

[IMG]http://www.hankstexasgrill.com/misc/cms/item_uploads/Boneless%20Wings_236.bmp[/IMG][IMG]http://www.hankstexasgrill.com/misc/cms/item_uploads/onion%20rings_963.jpg[/IMG][IMG]http://www.hankstexasgrill.com/misc/cms/item_uploads/Chips%20N%20Salsa_163.jpg[/IMG]
[IMG]http://www.jeremylatham.com/images/cold-beer.jpg[/IMG]

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Anonymous
June 12, 2007 at 10:13 pm

[QUOTE=sylvia’85]Hey Dan, the NCBI links wouldn’t come up for me. What were the titles of the articles you found?

Ali- I just sent you a PM:)[/QUOTE]

Abstract

To determine the involvement of the corticospinal tract in Guillain-Barré syndrome (GBS), we examined central motor conduction in patients with GBS-like symptoms and hyperreflexia using a magnetic stimulation technique. The subjects were 3 patients who exhibited ascending muscle weakness 2-4 weeks after preceding infections. Deep tendon reflexes were exaggerated in all four limbs of the 3 patients. The results of cerebrospinal fluid examinations revealed protein elevation without pleocytosis. The serum anti-GM1 antibody titer was elevated in 2 patients. The results of nerve conduction study revealed axonal motor neuropathy and normal F-wave conduction. Central motor conduction time (CMCT) in patients with hyperreflexia was significantly delayed compared to that in patients with GBS and areflexia (p < 0.001), and the delayed CMCTs were significantly improved in the recovery periods (p < 0.001). Although hyperreflexia is a controversial symptom in patients with GBS, these findings indicate that there is functional corticospinal tract involvement in patients with a GBS variant. Copyright © 2001 S. Karger AG, Basel -------------------------------------------------------------------------------- Author Contacts Takao Mitsui, MD First Department of Internal Medicine, School of Medicine University of Tokushima, Kuramoto 3-18-15 Tokushima 770-8503 (Japan) Tel. +81 886 33 7120, Fax +81 886 33 7121, E-Mail [email]tmitsui@clin.med.tokushima-u.ac.jp[/email] --------------------------------------------------------------------------------